Although many drug treatment programs claim to “deal with your feelings.” Chapman House actually developed the process! You WILL know how to deal with your feelings when you complete our drug rehabilitation program in Orange County.
Chapman House Treatment Centers provide a safe and comfortable residential detox. Our professional staff directly monitors our detox patients every 20-minutes. Simple, our program works! It is the only program of its kind that actually deals with the emotional state of an addicted individual.
Our partial hospitalization program, or PHP, is a highly structured outpatient program where patients receive treatment at least 5 days per week for no less than 6 hours per day.
Chapman House Treatment Centers provide a wide variety of outpatient services in addition to all of our primary drug treatment programs in Orange County.
Our residential treatment, also known as inpatient treatment, is a truly comprehensive and modern rehabilitation program
The least restrictive of our treatment programs, Intensive Outpatient or IOP, consists of a minimum treatment of three days a week for three hours per day.
Dual-diagnosis is a term for when someone experiences a mental illness and a substance abuse problem simultaneously.
You are a person afflicted with a disease or mental health issue or both. Therefore, you will never feel judged by our professional staff. Many of the staff here are in recovery themselves. Judgment STOPS at our door.
Next, what many people don’t realize, is that once a person stops drinking and/or using addictive substances; they no longer have an alcohol or drug problem, they have a LIVING problem! That’s where Chapman House excels with drug rehabilitation programs near you. We don’t sit around reminiscing about the “good (bad) ol’ days,” but rather, we teach people how to manage their lives. Whether its drugs, alcohol, mental-health issues or a combination, Chapman House can and will, help you to deal with your issues, and get your life back with the drug treatment programs you need in Orange County!
You WILL know how to deal with your feelings when you complete our program
Although many programs claim to “deal with your feelings.” Chapman House actually developed the process!
The Centers for Disease Control (CDC) estimates that approximately 35% of Americans receive less than seven hours of sleep daily and one in four Americans will develop acute insomnia at least once every year.
Ambien is a prescription-only sleep aid belonging to the non-benzodiazepine, sedative hypnotic class of sleep medications known as “z-drugs.” It is approved by the FDA (Food and Drug Administration)to treat insomnia characterized by difficulty falling asleep. Due to its habit-forming nature, it should not be taken long term (> 6 weeks) due to the potential for dependence and addiction.
Originally developed as a safer alternative to benzodiazepine drugs such as Xanax (alprazolam), Valium (diazepam) and Ativan (lorazepam), Ambien carries significantly less risk of seizures upon withdrawal as compared to benzodiazepines, however seizures can still occur. It was also marketed as being less addictive than benzodiazepines as it takes longer to develop tolerance and dependence. Research has shown that Ambien carries less potential for rebound insomnia upon discontinuation than benzodiazepines. While safer than benzodiazepines, Ambien still carries a significant risk of addiction and dependence, even in those without a history of substance abuse.
Due to these side effects, it is imperative that Ambien be taken immediately before going to sleep.
Listed below are real examples of when an individual did not go to sleep after taking Ambien
Because of Ambien’s strong amnesiac effects, these individuals remembered nothing about the events.
If Ambien is taken longer than the recommended maximum of six weeks, dependence and tolerance can develop, even in those who began taking the drug for a legitimate medical purpose. Tolerance occurs when the dose of Ambien that helped an individual get to sleep is no longer effective, necessitating an increase in the dose to achieve the same effect. Dependence can also occur, where sleep is difficult to achieve without Ambien and withdrawal symptoms are experienced upon discontinuation. Discontinuation may result in withdrawal symptoms such as depression, anxiety, sweating, and abdominal/muscle cramps. Withdrawal symptoms will generally peak 3-5 days after discontinuation and subside after 1-2 weeks.
It is important to note that while tolerance and dependence will develop in those who take Ambien for a prolonged period, this does not imply Ambien abuse, as dependence and tolerance will occur regardless of whether the drug is being abused. Some individuals will intentionally abuse the drug, either taking more than what was prescribed, obtaining the drug illegally without a prescription and/or taking it in a manner not consistent with how it was prescribed such as crushing or snorting it.
While Ambien was originally developed as a less addictive alternative to benzodiazepines, a growing body of evidence has suggested it may be much more addictive than previously thought, with the potential for abuse and addiction being as severe as its benzodiazepine counterparts. Research has shown that both Ambien and triazolam, a benzodiazepine also FDA-approved for the treatment of insomnia, showed a similar degree of “drug-liking,” a measurement of a drug’s potential for abuse.
Ambien addiction is a serious problem; those seeking addiction treatment require the assistance of a healthcare professional such as an addiction specialist. In most cases, the goal is discontinuation of Ambien to eliminate reliance on the drug to fall asleep. In order to accomplish this goal, a taperprogram is often used, where the dose of the drug is decreased slowly over time. By tapering off Ambien, the body can slowly adjust to reductions in dose. This is required because reducing the dose too quickly can result in withdrawal symptoms such as insomnia, anxiety, sweating and drug-craving.
Users may attempt to quit Ambien by going “cold-turkey,” where they suddenly and completely stop taking the drug after prolonged use. It is critical to note that this practice is extremely dangerous, as seizures have been reported upon discontinuation of high-dose Ambien. Rather, a safer and more effective way to detox from Ambien is to consult with a qualified addiction specialist in a treatment center who will develop an individualized taper regimen to slowly wean off the drug, minimizing withdrawal symptoms.
How long a taper will last is highly dependent on the individual and includes factors such as the Ambien dose, length of Ambien use and the ability to tolerate withdrawal symptoms upon dose-reductions.
The important thing to remember is that tapering is not a race; every individual will respond differently and there is no advantage to getting off faster.
Even with a slow taper, withdrawal symptoms may be experienced. Below are some drugs that may be used to manage the withdrawal symptoms experienced when discontinuing and/or tapering Ambien.
Some treatment options for Ambien addiction may involve the use of a long-acting benzodiazepine such as Valium to taper. It may seem strange that a benzodiazepine would be used in the treatment of Ambien addiction, considering it was discussed earlier that benzodiazepines are more dangerous than Ambien. The rationale for their use is due to the drugs’ half-life, a measure of how long a drug stays in the body. In general, the longer the half-life of a drug, the longer it takes until withdrawal symptoms start and the less severe they are. Ambien has a half-life of approximately 2.6 hours while Valium has a half-life between 20-50 hours depending on age. Because Valium and Ambien work similarly on a chemical level, Valium can be used to “replace” the Ambien, reducing the intensity of withdrawal symptoms and their time to onset.
Barbiturates are a class of drugs that act as central nervous system depressants. They are used for the treatment of seizure disorders, anxiety, insomnia and headaches. While they are still used as effective agents for the treatment of headaches, their use has largely fallen out of favor for treating seizures, anxiety and insomnia, as newer, safer sedative hypnotics have been developed such as benzodiazepines.
Barbiturates are available in pill form, suppositories and intravenous solution for injection. Also known as barbs, Christmas Trees, Goof Balls, and Red Devils, barbiturates are notable for their addictive potential and narrow therapeutic index.
Some common barbiturates are listed below:
** Only the butalbital component of this drug combination is a barbiturate
Barbiturates mimic a natural chemical in our brain called GABA (gamma-aminobutyric acid), the primary inhibitory neurotransmitter of the central nervous system. By mimicking GABA, barbiturates are essentially “stepping on the brakes” in our brain by slowing down brain activity. While this makes them effective drugs for anxiety and seizures (overactive brain activity), slowing brain activity down too much can result in coma and death.
As central nervous system depressants, the effects of barbiturates resemble that of alcohol and benzodiazepines such as Xanax (alprazolam) and Valium (diazepam). The table below lists some of the psychological and physical effects of barbiturates.
Psychological EffectsPhysical EffectsEuphoriaShallow breathingMemory impairmentWeak pulseLowered inhibitions, impaired judgementClammy skinDizzinessDilated pupilsSedationComa and death
A major reason why barbiturates are so dangerous is due to their narrow therapeutic index. The therapeutic index of a drug is a measure of a drug’s safety. It is essentially a “window” between a dose low enough to be effective and a dose at which it becomes dangerous.
Because of their narrow therapeutic index, it is incredibly easy to overdose on barbiturates compared to other central nervous system depressants such as benzodiazepines. During the 1960s and 1970s, when barbiturates were at the peak of their use in the United States, they were responsible for the deaths of many celebrities, including:
While dangerous on their own, the potential for deadly overdose increases dramatically when barbiturates are combined with other central nervous system depressants such as alcohol and opioids.
As a drug of abuse, many barbiturate users will take the drug in higher doses than prescribed or obtain it illegally to feel the “high” associated with their use. It is also a popular drug to soften the crash associated with cocaine and methamphetamine.
Many users prefer the use of short-acting barbiturates such as pentobarbital and secobarbital, which are particularly dangerous because withdrawal symptoms such as seizures begin soon after the effects of the drug wear off.
Psychological symptoms of barbiturate abuse involve degradation in the user’s mental health, and may include the following:
Physical symptoms of barbiturate abuse include the following:
Tolerance to barbiturates develops quickly. Tolerance is when a higher dose is required to exert the same effect that was felt at a lower dose. Even after 1-2 weeks of taking barbiturates, higher doses of barbiturates may be required to exert similar effects, which may lead to users increasing the dose and exposing themselves to dangerous side effects.
Both physical and psychological dependence to barbiturates may develop upon extended use.
Psychological dependence to barbiturates develops when users must take it to feel normal and experience craving for the drug when the effects wear off. Physical dependence to barbiturates can also develop, where the body adapts to the use of the drug. Physical dependence will always develop when barbiturates are used long term, even in those without a history of substance abuse.
Withdrawal from barbiturates can be life-threatening if not managed appropriately. When the body becomes dependent on barbiturates to function normally, stopping the drug results in withdrawal symptoms. Withdrawal symptoms can be both psychological or physical and are listed in the table below.
Psychological Withdrawal Symptoms
Physical Withdrawal SymptomsDrug cravingSeizuresAnxietyTremorsAgitationFeverInsomniaNausea and vomiting
The most dangerous withdrawal symptom of barbiturates is seizures, as they can be deadly if not controlled. How long it takes until seizures develop depends on the half-life of the barbiturate being abused. The half-life of a drug is a measure of how long it stays in the body. Barbiturates with a short half-life are eliminated quickly, and seizures can develop as soon as 16 hours following the last dose, lasting up to five days. Barbiturates with a longer half-life stay in the body longer and take longer until withdrawal symptoms appear.
Treatment involves managing both the psychological and physical effects of barbiturate addiction. While the goal of treatment is to discontinue use or limit use only to the degree necessary to treat the problem, it is imperative that barbiturates not be discontinued suddenly (“cold turkey”), as it exposes the individual to lethal seizures.
To control the immediate withdrawal symptoms associated with barbiturates, a barbiturate with a long half-life, such as phenobarbital, is often used to replace the barbiturate being abused. Because phenobarbital stays in the body for a long period of time, it allows withdrawal symptoms to stay under control while the dose of the drug is slowly decreased (tapered).
Some clinicians may choose to switch to a long-acting benzodiazepine such as Valium (diazepam) to taper, as it is safer than barbiturates.
It is important to keep in mind that attempting to detox from barbiturates by suddenly discontinuing (cold turkey) is incredibly dangerous and should not be attempted. A much safer and more effective way to detox from barbiturates is to seek the help of a qualified addiction specialist that will help choose an individualized plan to manage addiction.
Cocaine is a stimulant drug abused for its euphoric effects. While it serves a legitimate medical purpose as a topical anesthetic for surgeries, it is more commonly encountered as a powerful and addicting illegal drug.
Usually sold as a white powder or rock-like substance (crack cocaine), users will snort, smoke or inject cocaine to feel its energizing, euphoric effects. In an attempt to increase profits, dealers will often dilute cocaine with other substances such as talc, baking soda, cornstarch, or drugs such as methamphetamine and fentanyl.
Popular street names for cocaine include “coke,” “blow,” “crack,” “snow,” and “rock.”
Cocaine use remains a major problem in the United States. The National Institute on Drug Abuse shares the following statistics regarding cocaine use:
Cocaine VS Crack – What’s the Difference?
Cocaine comes in two main forms, powdered cocaine, also known as “cocaine hydrochloride,” and crack cocaine. Pure powdered cocaine is a white, crystalline powder that can be snorted and injected. It cannot be smoked in this form because it is inactivated by high temperatures such as exposure to a flame. To make the drug smokable, it must be converted into a form known as “crack.”
Crack cocaine is made by mixing cocaine powder, baking soda and water, then boiling it until a solid rock-like substance is formed.
The table below highlights the differences between powdered cocaine and crack cocaine.
While all forms of cocaine are dangerous, crack cocaine’s low cost makes it particularly dangerous because it is more accessible. Due to its low price, ease of use and intense effects, crack is often considered to be more addicting than its powdered cocaine counterpart.
Most users choose to snort powdered cocaine, however it can be injected or smoked as well.
Cocaine works by stimulating our brain’s “reward pathway,” a primitive part of our brain responsible for reinforcing positive stimuli necessary for survival. For example, when we are hungry and eat food, our reward pathway “rewards” us for eating by releasing dopamine, leading to a pleasurable and satisfying feeling. Cocaine hijacks this system and enhances its effect, tricking our brains into rewarding us for cocaine use.
Cocaine abuse is associated with significant side effects. Short term effects of cocaine may include the following:
All amounts of cocaine can produce these effects, however the higher the dose, the more likely and severe these effects will be. Injecting and smoking will produce stronger effects that appear faster, while snorting will produce more mild effects that may taker longer to set in.
Long term effects of cocaine use include the following and will vary depending on how the drug is taken:
Since cocaine is often diluted with other ingredients, including dangerous drugs such as fentanyl, its true strength is nearly impossible to determine. This opens up the possibility of a dangerous overdose.
Overdose on cocaine is life-threatening and may involve the following symptoms:
No antidote for cocaine exists – treatment for overdose is often supportive and will involve treating the symptoms.
Cocaine is a powerfully addictive drug; users can become addicted even after a single use. Repeated use of the drug may lead to addiction.
Signs and symptoms of addiction may include the following:
When used repeatedly, tolerance will develop to the euphoric effects of cocaine, where more of the drug must be taken to achieve the same effects.
Long term use can cause a user to become dependent, where they require the drug to feel normal. If dependent, the user will suffer withdrawal symptoms if the drug is not taken.
Withdrawal symptoms of cocaine include:
There are no notable physical withdrawal symptoms, however this does not make cocaine easy to quit, as the drug craving associated with cocaine withdrawal can last months after quitting.
Since withdrawal from cocaine is not physically dangerous, addiction treatment does not require admittance to a treatment center or rehab, however a user may still benefit greatly from the support of a therapist specializing in substance use disorders.
Fentanyl is an extremely potent and dangerous synthetic opioid drug. As a prescription drug, it is used for severe pain in those who have built tolerance to other opioids. The term “synthetic opioid” means that it is completely man-made in a laboratory; no form of fentanyl exists in nature. As an opioid pain reliever, fentanyl was designed to be 50 to 100 times more potent than morphine. Due to its strength, the risk of overdose is incredibly high. Because it is relatively cheap and easy to manufacture, fentanyl is often added to other illegal opioids such as heroin to increase their strength. At Chapman House, we understand just how dangerous this drug can be for your life. Getting help from one of our Fentanyl rehab programs can bring your life back into focus. With our Fentanyl addiction treatment, you’ll have caring health professionals who can guide you every step of the way during recovery.
Several versions of fentanyl exist, including prescription products such as a transdermal patch, lollipop, solution for injection as well as illegal street powder and blotter paper.
An opioid epidemic has swept the United States, involving widespread use of both prescription and illegal opioids. From 1999 – 2017, opioids have been responsible for approximately 400,000 deaths, with nearly 130 deaths every day being attributed to opioids.
Fentanyl is a relatively new player in the opioid epidemic. While fentanyl has been available as a prescription product in the United States since the 1960s, its use as an illicit drug has only started to rise since the early 2010s. It is believed that most illegally produced fentanyl is manufactured in China and smuggled into the United States through Mexico.
A recent report from the Centers for Disease Control and Prevention clearly illustrates the dangers of fentanyl. In 2016, the number of deaths involving fentanyl was estimated to be 18,335 compared to only 1,663 in 2011. This represents more than a ten-fold increase in deaths involving fentanyl from 2011 to 2016.
Because drug dealers will often add fentanyl to heroin, the rise of heroin-related deaths has grown due to fentanyl’s popularity. As shown on the graph above, heroin-related deaths have increased rapidly since 2011.
Fentanyl is an opioid agonist, meaning it binds to opioid receptors in the central nervous system. When fentanyl binds to opioid receptors, it blocks pain signals, making them invaluable tools for alleviating pain. Despite its use a legitimate painkiller, it also has the potential to make the user feel euphoric, which is why it is so addicting.
Opioids like fentanyl also cause a significant amount of side effects. Listed below are some of the most common side effects of fentanyl.
The most dangerous side effect of fentanyl is respiratory depression. Respiratory depression is when breathing becomes shallow and ineffective. If too much fentanyl is taken, respiratory depression can be so severe the user stops breathing completely.
While all opioids carry the risk of respiratory depression, fentanyl’s high potency significantly increases the risk of overdose because only a small amount is required to cause respiratory depression.
Fortunately, there is a product that can reverse the deadly respiratory depression associated with a fentanyl overdose – Narcan. Narcan is a nasal spray containing the drug naloxone, an opioid antagonist. It works by removing fentanyl from the user’s system to restore breathing.
Narcan is available without a prescription in California. If you or someone you know is addicted to fentanyl, having Narcan on-hand can mean the difference between life and death.
It is important to know the signs of fentanyl overdose, which can include:
Because fentanyl is incredibly strong at even small doses, many users will take it when they no longer feel the effects from weaker opioids such as heroin. This can easily lead to continued abuse and addiction, which is something Chapman House is here to help prevent or provide support with in recovery through our Fentanyl addiction treatments.
The length of time until the effects of fentanyl can be felt (onset) and how long the effects last (duration) depend on how it is taken.
OnsetDurationIntravenous injectionInstantly30 min – 1 hourSmokedInstantly30 min – 1 hourSkin patch6 hours72 hours
Like all opioids, tolerance can develop to fentanyl, where more of the drug is required to feel the same effects. This leads to users taking more fentanyl to feel the same effects and further exposing themselves to dangerous side effects.
Dependence on fentanyl can also occur when it is taken long term. Dependence is when the body requires the drug to function normally. Once a user is dependent, withdrawal can occur if they do not take the drug. However, even once dependent, there is always a chance to overcome your addiction, especially with help from caring professionals at Chapman House’s Fentanyl rehab programs.
Withdrawal from fentanyl, while usually not life-threatening, can be incredibly painful and uncomfortable. Most users who are addicted to fentanyl will continue taking it not only to feel high but also avoid withdrawal.
Some withdrawal symptoms of fentanyl are listed below.
Withdrawal symptoms will start within 24 hours of the last fentanyl dose, peak within 1-3 days and subside over the following 1-2 weeks.
Since this is an opioid, Fentanyl addiction treatment will follow typical guidelines for opioid use disorders.
While the goal of addiction treatment is complete detoxification from the drug, the immediate goal is to control withdrawal symptoms. This can be accomplished by switching to a long-acting opioid such as methadone to control withdrawal symptoms. Some practitioners may recommend the use of newer drugs for opioid dependence such as Suboxone or Subutex.
Once fentanyl is replaced with a safer opioid, the dose is slowly tapered (decreased) over time until it can be safely discontinued. How long the tapering period will last is dependent on many factors including how long the user was abusing fentanyl, the dose of fentanyl being abused and the ability to tolerate dose reductions during the tapering period. If you have been struggling with your own addiction, let the experts at Chapman House help you along the road to recovery through our Fentanyl rehab programs!
Heroin is an illegal opioid drug abused for its intense euphoric effects. While it can be smoked or snorted, most users choose to inject heroin intravenously. Following injection, users immediately experience an intense, pleasurable “rush” followed by hours of a relaxed, trance-like state known as a “nod.”
Heroin is often seen as a white or brown powder; its color and consistency may vary depending on impurities added to it. “Black tar heroin” is a sticky, gummy form of heroin resembling roofing tar that is commonly seen in areas of the United States west of the Mississippi River.
Unlike a prescription opioid such as hydrocodone or oxycodone, which is legal to possess with a prescription, all forms of heroin are illegal.
Heroin is derived from morphine, a natural alkaloid of the opium poppy plant. Most illicit opium originates in Afghanistan, where it is grown as a cash crop and exported to Europe and the United States. The opium is refined into morphine, then converted into heroin through chemical processes.
The conversion of morphine to heroin makes it stronger and facilitates more rapid movement across the blood-brain-barrier, allowing the effects to hit faster.
Heroin may be “cut”, or diluted, with dangerous impurities to bulk up its weight. Not only is this dangerous because the user can never be sure how much heroin they are taking, but the impurities themselves can be dangerous. Some impurities that are commonly added to heroin include:
Of all the impurities, fentanyl is perhaps the most dangerous. Fentanyl, like heroin, is an opioid, except significantly stronger. Only small amounts of fentanyl are required to cause an overdose, making heroin diluted fentanyl particularly deadly.
Heroin and the Opioid Epidemic
The United States is currently experiencing an “opioid epidemic.” Over the past 20 years, opioids have contributed to an increasing number of deaths countrywide. The Centers for Disease Control (CDC) estimates that between 1999 and 2017, opioids have been responsible for more than 700,000 deaths.3
The number of overdose deaths due to heroin has risen dramatically from 1,960 in 1999 to 15,482 in 2017, nearly an eight-fold increase.
Heroin is a potent opioid agonist that binds to opioid receptors in the central nervous system. After injection, heroin enters the brain and latches on to opioid receptors, causing an intense, pleasurable feeling known as the “rush.”
When heroin is used repeatedly, opioid receptors become less sensitive to its effects. This results in tolerance, where a higher dose of the drug is required to feel the same effect.
In addition to the feeling of euphoria, heroin is also associated with a significant amount of side effects, which include:
Respiratory depression is characterized by slow, ineffective breathing that can be fatal if not treated quickly.
Someone who has overdosed on heroin is potentially at risk of death due to respiratory depression. It is important to be able to identify the signs and symptoms of heroin overdose.
If someone is suspected to have overdosed on heroin, emergency services should be called. The drug Narcan is used to reverse heroin overdose. Narcan is an opioid antagonist available as a nasal spray, working within minutes to reverse respiratory depression. It is available without a prescription in most states, including California.
Because heroin is illegal, any use is considered abuse as there is no medically accepted reason why someone should be taking it. People who use heroin repeatedly can quickly spiral down the path of addiction, where their life revolves around obtaining and taking the drug.
The following signs may signal addiction to heroin:
Over time, tolerance will develop to heroin, where the user takes more to achieve the same effect. Long term use will make the user dependent, meaning they require it to feel normal. Once dependent, a user will suffer painful withdrawal symptoms soon after the effects wear off.
Heroin withdrawal is an incredibly painful experience; so painful in fact that most users will continue taking heroin not only to experience the high, but also in fear of withdrawal. This is what makes heroin so difficult to quit, not only are the effects addicting, but stopping is painful.
Symptoms of heroin withdrawal are listed below:
Treatment of heroin addiction marks the first step towards the restoration of a user’s physical and mental health. The end goal of addiction treatment is complete discontinuation of heroin use and prevention of use in the future.
While some may attempt to quit “cold turkey” by suddenly and completely stopping heroin, this is associated with extreme withdrawal symptoms; most people are unable to cope with the severity of withdrawal and continue using it to alleviate withdrawal. It is recommended that heroin users admit themselves to a treatment center for drug detox where they can receive the help they need.
There are several options for the treatment of heroin addiction. Among the most common is opioid replacement therapy (ORT) with methadone or Suboxone.
Drug addiction is a difficult challenge to overcome, with heroin ranked as one the most difficult drugs to quit. Drug addiction specialists are available to keep users safe and mitigate the painful symptoms of withdrawal along the process of recovery.
The term inhalant refers to any volatile substance that can be inhaled to produce a psychoactive effect. In most cases, inhalants are legal, household products that can be bought at any warehouse store. Despite their seemingly innocuous nature, these chemical vapors can be extremely dangerous, some even causing death or permanent brain damage the first time they are abused.
The National Institute on Drug Abuse estimates that 9.3% of Americans 12 years old and older have tried an inhalant at least once in their life.
Typical inhalants may include the following:
Street names for inhalants include “air blast,” “bang,” “huff,” “dust,” and “whiteout.”
Inhalants are unique in that they are more popular among younger teens compared to older teens and adults. In fact, the National Institute on Drug Abuse’s annual Monitoring The Future survey of 8th, 10th and 12th graders found inhalant abuse more popular among 8th graders than any other group.
Several factors may contribute to inhalants’ popularity among young teenagers:
Since inhalants are in a gaseous or aerosol form, they are breathed in through the mouth or nose into the lungs. When the inhalant reaches the lungs, it crosses into the bloodstream and exerts its effects almost instantly.
The high associated with inhalants usually only lasts minutes. Many people continue to inhale the drug in an effort to continue the high.
Inhalants can be taken in a variety of ways including:
How Do Inhalants Work in the Brain?
Inhalants are a broad category of drugs with varying mechanisms of action, however most inhalants are central nervous system depressants similar to alcohol and benzodiazepines. One particularly inhalant, toluene (an ingredient in paint thinner), has been shown to increase dopamine in the brain. This mechanism is similar to how cocaine works. Nitrites, also known as “poppers” or “snappers,” work by dilating blood vessels to produce a lightheaded effect.
Because all inhalants are gaseous, they displace oxygen in the lungs, leading to deadly oxygen deprivation.
Poppers are a particularly popular class of inhalants called nitrites – drugs that dilate blood vessels to produce a fast-acting sensation of relaxation accompanied by dizziness and lightheadedness. They can be bought online, at bars, or gas stations. Some common nitrites contained in poppers include:3
Supplied as a small vial containing the nitrite in gaseous form, when the cap is removed the drug is quickly inhaled through the mouth or nose.
Poppers are associated with a significant amount of side effects including eye damage, increased heart rate, migraines and fainting.
Poppers are currently illegal in the United States.
Depending on which type of inhalant has been used, it may produce any number of the following effects:
Since inhalant use cuts of oxygen, an essential component to a health body and nervous system, many of the long term effects of inhalants involve problems with cognitive abilities or senses. Some of the long term effects include:
In high enough doses, most inhalants will produce anesthesia, or a loss of sensation often accompanied by unconsciousness. If the dose is too high, the user may slip into a coma and die.
Since many inhalants are very concentrated, it only takes a small amount of the drug to cause an overdose.
Inhalant abuse can cause death even after the first attempt. A major cause of death due to inhalant abuse is Sudden Sniffing Death Syndrome. Since inhalants can cut off oxygen, many users die from oxygen deprivation.
Inhalants can also make the user more sensitive to natural chemicals that increase heart rate such as adrenaline. When adrenaline is released to make the heart beat faster, the heart can overreact and end in a heart attack.
Since the effects of inhalant abuse are usually short-lived, it may be difficult to detect inhalant abuse while it is being abused. Nevertheless, the following signs may be signal inhalant abuse:
Treating Inhalant Addiction
Many users may come find the pleasurable feelings associated with inhalant use difficult to resist and may develop an addiction to them.
While users are unlikely to develop physical dependence on inhalants, their addiction may feature psychological dependence. These users may find it difficult to resist urges to use the drug and rely on it to feel normal.
An addiction specialist can help those suffering from inhalant addiction and provide a range of services to support both the user’s physical and mental health.
Behavioral therapies for addiction may be helpful to:
Initial treatment for addiction can start in one of two settings – inpatient or outpatient.
Inhalant abuse can cause long-term damage or death in a very short period of time, therefore it is imperative that inhalant addiction be managed as soon as possible.
Methamphetamine, commonly known as “meth”, is a highly addictive stimulant drug that affects the central nervous system. While it is available as a prescription drug under the brand name Desoxyn® indicated for the treatment of attention deficit hyperactivity disorder and obesity, it is more commonly encountered as an illegal and dangerous street drug. Also known as blue, ice, crystal and speed, meth takes the appearance of a white, odorless, bitter-tasting crystalline powder that can be smoked, insufflated (snorted) or injected. When smoked or injected, the high from methamphetamine is felt instantly, peaks within 15 minutes and typically lasts 4-8 hours.
According to a 2017 survey by the National Survey on Drug Use and Health, approximately 1.6 million Americans had reported using methamphetamine in the past year, with the average age of a first-time user being 23.3 years old. While methamphetamine is accessible anywhere in the country, its use is significantly more prevalent in the western and midwestern United States.
The Drug Enforcement Agency (DEA) has identified the primary source of large-scale methamphetamine production originating from Mexican drug cartels, which smuggle the drug across the United States-Mexico border.5 Meth is also produced in the United States as well, where users and/or distributors synthesize meth in clandestine “meth labs.” Far from pristine, meth labs use crude household products to manufacture meth in filthy conditions. Below are some of the ingredients used:
Possession of these ingredients should serve as a red flag to the possibility of meth production and/or use, particularly excessive purchasing of pseudoephedrine. In 2005, the Combat Methamphetamine Epidemic Act was passed to curb meth production. The Act mandates that retailers require photo identification upon purchase, track purchase history and limit the sale of pseudoephedrine-containing products.
Meth works by flooding the brain with dopamine, our body’s natural “feel-good” chemical, as well as norepinephrine, a chemical associated with arousal and alertness. As a result, users often experience intense euphoria, hyper-confidence and a feeling of never-ending energy.
Psychological symptoms of methamphetamine intoxication include:
Physical symptoms of methamphetamine intoxication include:
While under the influence, users may feel capable of anything and often engage in high-risk behavior such as gambling, criminal activity and unprotected sex.
Due to its long-lasting stimulant properties, meth users may go several days without sleep. During this period of no sleep, known as “tweaking,” the user becomes increasingly paranoid, aggressive and prone to mood swings as the lack of sleep wreaks havoc on their mental health.
The stages of methamphetamine abuse often follow a predictable “binge and crash” pattern outlined below:
Meth addicts often exhibit a characteristic appearance.
Long term effects of meth abuse can result in profound mental changes such as paranoia, psychosis and depression. Because meth addicts will often go days without eating and neglecting person hygiene, many addicts develop “meth mouth,” characterized by severe tooth decay. A 2015 study found that out of 571 meth users, 96% had cavities, 58% had untreated tooth decay and 31% had at least six missing teeth.
Many meth users choose to inject the drug intravenously, causing the effects to be felt instantly. This is perhaps the most dangerous method of taking meth, as research has shown that injecting a drug leads to addiction the fastest. In addition, the use of dirty needles can result in skin and soft tissue infections as well as heart valve infections such as endocarditis. Many users also share needles, drastically increasing the risk of contracting blood-borne diseases such as HIV and hepatitis C. The CDC estimates that intravenous drug users accounted for approximately 9% of HIV cases in 2016.
While these are general timelines, the length of each withdrawal phase varies for each individual. Users who have been abusing meth for a longer period can expect withdrawal to last longer, sometimes up to months until complete recovery.
Addiction treatment for meth is notoriously difficult, as there are no FDA-approved treatments. Unlike drugs such as opioids and benzodiazepines, withdrawal from meth is not likely to be physically dangerous, though that does not mean it is any easier to detox from.
The immediate goal of meth detoxification is complete cessation of meth and control of withdrawal symptoms. Some health care practitioners may choose to use benzodiazepines such as Xanax to control the anxiety and aggression associated with withdrawal as the body acclimates to functioning without meth. Treatment will primarily focus on behavioral therapies to reverse the psychological damage done by meth. These therapies may include counseling sessions and group therapy as part of the rehabilitation process.
Research has been conducted on the use of naltrexone, a drug commonly used to treat opioid and alcohol addiction, in the management of meth addiction. A 2015 study9 found that naltrexone significantly reduced meth craving following detoxification. It was also found to dull the pleasurable feelings associated with meth. With drug craving being a core symptom of meth addiction that persists long after withdrawal symptoms subside, naltrexone may have utility in preventing relapse following successful detoxification from meth.
Treatment of meth addiction is a difficult process that requires the support of a qualified healthcare professional. While some attempt to quit meth on their own without help, it is significantly more effective to enroll in a treatment program that can help manage withdrawal symptoms and build strategies to prevent future use.
OxyContin is a prescription-only opioid used in the management of moderate to severe pain. It is a special formulation of the drug oxycodone designed to slowly release the drug over an extended period of time, providing around-the-clock pain relief. Compared to other opioids which may last 6-8 hours, OxyContin provides sustained delivery of oxycodone for up to 12 hours to treat chronic pain.
Because it slowly releases the drug, the dose of oxycodone contained in OxyContin is significantly higher than other oxycodone product such as Percocet. OxyContin also contains pure oxycodone, whereas Percocet contains a combination of oxycodone and acetaminophen (the active ingredient in Tylenol).
Due to its high content of “pure” oxycodone, OxyContin carries a significantly higher abuse potential than other oxycodone products. Many users will attempt to crush the pill, bypassing the controlled-release mechanism in order to get the entire amount of oxycodone into their system faster.
Street names for OxyContin include oxys, OC’s, oxycotton, hillbilly heroin, and berries.
OxyContin was introduced to the US market in 1996 by Purdue Pharma. The manufacturer’s aggressive marketing campaign systematically downplayed the drug’s addiction potential, even training its sales representatives to tout the addiction potential as “less than one percent.”
Evidence based research has since shown OxyContin to be extremely addicting. Many states, most recently California, have brought lawsuits against the drug-maker for its deceptive sales practices.
OxyContin contains a large dose of oxycodone that is released over 12 hours to provide long term pain relief. This makes the drug incredibly useful for those suffering from chronic, severe pain. Medical professionals may issue an OxyContin prescription for the following:
OxyContin is an opioid agonist that binds to opioid receptors in the central nervous system. By binding to opioid receptors, OxyContin blocks pain signals, which is why it is used as a painkiller. While effective at treating pain, it also makes the user feel a strong sense of euphoria, making it incredibly addicting even for those without a history of substance abuse.
OxyContin is associated with a significant amount of side effects, which can include:
Respiratory depression is defined as slow and shallow breathing; it is the most dangerous side effect of OxyContin. If OxyContin is taken in too large of a dose, as is common in those who abuse the drug by crushing it, respiratory depression can be fatal.
In cases of OxyContin overdose, the drug Narcan can be used to reverse respiratory depression. Narcan contains the opioid antagonist naloxone in the form of a nasal spray. When used correctly, it can save the life of someone who has overdosed. In most states, including California, it is available without a prescription.
Because OxyContin makes users feel incredibly euphoric, its use is associated with a significant potential for abuse and addiction. Even those who begin taking OxyContin for a legitimate medical reason are not immune to the addictive potential of OxyContin. Those who seek out the drug solely to abuse it will often crush the pill, breaking the time-release mechanism and releasing the entirety of its contents. Users will often attempt to snort or even inject the contents to feel the high immediately.
Injection of OxyContin is particularly dangerous, as it immediately introduces the drug into the bloodstream. The high experienced from injection is similar to heroin, which is why OxyContin is sometimes referred to as “hillybilly heroin.”
Users who inject OxyContin not only expose themselves to the dangers of an overdose, but sharing needles is associated with a significant risk of blood-borne diseases such as HIV and hepatitis C.
When OxyContin is repeatedly used, the body builds resistance to its effects, known as tolerance. Tolerance develops quickly to the euphoric feelings of OxyContin, causing users to take more of the drug to feel the same effects.
When used over a long period time, the body can become dependent on the drug, requiring it to function normally. Users can become both psychologically and physically dependent on Oxycontin. Psychological dependence is characterized by needing the drug to feel normal. Physical dependence is when the user suffers physical withdrawal symptoms if they do not take it.
Symptoms of withdrawal can be both psychological and physical. Some withdrawal symptoms are listed below.
When a user experiences withdrawal from OxyContin, they will often engage in extreme drug seeking behavior such as drug craving and lying or stealing from friends and family members to obtain more of the drug.
OxyContin addiction is a difficult challenge to overcome; the goals of treatment focus on both the physical and mental health of the individual.
Those admitted to an inpatient treatment facility will often be switched to a different opioid such as methadone or Suboxone to control withdrawal symptoms; this is known as “opioid replacement therapy” or “opioid substitution therapy.”
Methadone is a commonly used opioid for opioid replacement therapy, as it has a long half-life that provides sustained control of withdrawal symptoms. Some addiction specialists may recommend the use of newer therapies such as Suboxone or Subutex.
The dose of the replacement opioid is slowly decreased (tapered) over time, allowing the body to adjust to dose reductions until it can be safely discontinued.
Some users may attempt to suddenly discontinue OxyContin, known as “going cold turkey.” This is potentially dangerous, as seizures have been observed in those who suddenly discontinue high-dose opioids such as OxyContin.
It is highly recommended that anyone suffering from OxyContin addiction seek the help of a qualified addiction specialist who can help them with their substance use disorder and choose a therapy that fits their unique needs.
Stimulants are a class of drugs that increase the activity of the central nervous system. Some stimulants, such as Adderall and Ritalin, are prescription drugs used for the treatment of attention deficit hyperactivity disorder and narcolepsy. Other stimulants include illegal drugs such as methamphetamine, cocaine or ecstasy.
Stimulant drugs are one of the most abused classes of drugs in the United States, ranking 3rd behind marijuana and prescription drugs.1 The National Survey on Drug Use and Health reports that at least 14% of Americans have tried cocaine at least once in their life and at least 8.5% have tried an amphetamine such as Adderall without having a prescription.
While there are differences between each stimulant, stimulant abuse in general is associated with a short-lived, intense high, followed by a crash, where the user feels depressed and may crave the drug.
Cocaine is a naturally occurring alkaloid found in the leaves of the coca plant, indigenous to South America. While it is occasionally used as a topical anesthetic for surgeries due to its numbing effect, it is most often encountered on the street as a dangerous and addicting stimulant.
It is available on the street under several names such as “coke,” “blow,” “nose candy,” and “yayo,” usually as a white powder. Most users choose to snort it, however it is easily dissolved in water and injected into a vein. Powdered cocaine cannot be smoked however it can be converted into crack cocaine, a smokable form of the drug.
A special form of cocaine, known as “crack” or “rock,” is made by adding baking soda to cocaine and heating the mixture up.2 This forms a solid rock that can be smoked, often in a pipe called a “crack pipe.” Compared to powdered cocaine, crack cocaine is significantly cheaper.
In many ways, crack cocaine is even more dangerous than powdered cocaine. Its low cost makes it more accessible, and because smoking it causes the effects to be felt immediately, crack has been shown be more addictive than powdered cocaine.
Cocaine is associated with an intense high, where the user feels focused, confident and may engage in risky behavior such as stealing, gambling or unprotected sex. It also carries a significant amount of side effects, listed below:3
Long term use may be associated with the following:
How long it takes to feel the effects of cocaine and how long they last depend on the way it was taken. When cocaine is smoked or injected, the effects are felt almost instantly. When snorted, it may take minutes to feel the effects.
Amphetamines are a class of prescription stimulants used for several legitimate medical conditions. Some of the following medical uses of amphetamines are listed below:
Some of the most common amphetamine drugs include:
Methamphetamine, also known as “meth,” “crystal,” and “glass,” also belongs to the amphetamine class of drugs. While methamphetamine is available as a prescription drug to treat ADHD, it is most commonly seen on the street as an impure, rock-like substance that is smoked, snorted or injected.
Methamphetamine is an incredibly dangerous and addicting drug that can severely affect a user’s physical and mental health. Users will often abuse meth in a “binge and crash” cycle where they take the drug for days at a time, often going 3-4 days with no sleep. This is followed by a crash as they enter a state of depression and exhaustion.
Adderall, a prescription drug containing mixed amphetamine salts, is incredibly popular among college students. Because it can help users focus, it is often used to cram before big exams or term papers. In one survey asking 1,300 college students if they had used an amphetamine to help study, approximately 25% said they did, however only 8.9% of the students had a prescription for it.5
Methylphenidate is a stimulant drug similar to amphetamines. It goes under several brand names including Ritalin and Concerta, used to treat ADHD and narcolepsy. While not technically an amphetamine, its effects are virtually the same as amphetamine stimulants.
The signs and symptoms of stimulant abuse may vary from person to person. Individuals who are addicted to stimulants such as Adderall or Ritalin for study/work-related purposes may hide their addiction very well since they are likely to maintain success in work or academics. Some signs of stimulant addiction, however, are difficult to hide and may include:
Like most drugs of abuse, tolerance can develop to the effects of stimulants, where the user must take more in order to achieve the same effect. Dependence can also develop, where the user must take the drug to feel normal. Many college students who take stimulants such as Adderall or Ritalin illegally for a long period of time find it difficult to study without it.
Once a user is dependent on a stimulant they will experience withdrawal symptoms if they do not take it.
Withdrawal from stimulants is generally not physically dangerous, however coping with the drug craving experienced during withdrawal can be incredibly difficult.
Some symptoms of stimulant withdrawal include the following:
Treatment for addiction to stimulants such as cocaine, methamphetamine or prescription amphetamines will often focus on repairing the mental health problems caused by long term stimulant use.
In some situations, antidepressants are used to help alleviate the depression associated with stimulant withdrawal and treat underlying depression. Naltrexone, a drug usually used for opioid dependence, is also used to help with drug craving as an adjunct to behavioral therapies.
Substance abuse is a very difficult challenge to overcome. While stimulant withdrawal is not physically dangerous, it is highly recommended that a user seek help from a healthcare professional who specializes in substance use disorders.
Suboxone is the brand name of a drug combination containing buprenorphine and naloxone. It was approved by the Federal Drug Administration (FDA) in 2002 for the treatment of opioid dependence. Suboxone is available as sublingual films that are dissolved under the tongue (sublingual) or inside the cheek (buccal).
The Centers for Disease Control (CDC) has described the current pattern of opioid use in the United States as an “opioid epidemic.” Below are some of the CDC’s harrowing statistics regarding the opioid epidemic in the United States.2
The Substance Abuse and Mental Health Services Administration (SAMHSA) estimated that in 2017 approximately 2.1 million people in the United States had abused or were dependent on opioids.3 Many of those dependent on opioids are prescribed Suboxone to treat their dependence.
Not all prescribers can prescribe Suboxone. Under the Drug Addiction and Treatment Act (DATA), only qualifying prescribers may prescribe Suboxone after completing a special training program. To find a Suboxone prescriber near you, visit the SAMHSA buprenorphine practitioner locator.
Opioids are drugs that bind to opioid receptors in the brain. They are addicting because they mimic endorphins, our body’s natural “feel good” chemicals. There are three main types of opioids in terms of how they interact with opioid receptors: agonists, partial agonists and antagonists.
Addicting opioids such as heroin, morphine, hydrocodone, oxycodone and fentanyl are all opioid agonists, drugs that fully activate the opioid receptor. As the dose of an agonist increases, so does the effect.
Buprenorphine, one of the two ingredients in Suboxone, is a partial agonist. As the dose of buprenorphine increases, so does the effect, but only until a ceiling is reached. As shown on the graph below (orange is Suboxone), as the dose increases, the effect increases steadily until a plateau is reached. Once the plateau is reached, the effect remains the same even when the dose increases. This is an important property of buprenorphine, as a user is less likely to abuse it by taking higher doses. Once the plateau is reached it is impossible for the user to feel “higher” by taking more of it.
In addition to the opioid buprenorphine, Suboxone also contains the opioid antagonist known as naloxone. When naloxone is taken, it blocks the effects of opioids. If someone who is not dependent on opioids takes naloxone, there will be no effect. However, if someone dependent on opioids takes naloxone, they will immediately suffer painful withdrawal symptoms as the naloxone flushes the opioids out of their system.
This may seem very strange at first. Why would a drug used to treat opioid addiction contain an ingredient that can cause painful withdrawal symptoms? The reason is due to naloxone’s bioavailability. Bioavailability is the percent of a drug that reaches the blood stream to exert an effect. The bioavailability of naloxone is different depending on how it is taken.
As shown in the table above, when naloxone is dissolved under the tongue or inside the cheek (the way Suboxone should be taken), it has no effect. If naloxone is injected or snorted, it exerts its full effect as an opioid antagonist, immediately producing painful withdrawal in an opioid-dependent individual.
Because naloxone causes no effect when taken as prescribed but can cause painful withdrawal when it is abused, it is added to Suboxone to deter drug abuse and ensure it is taken exactly as prescribed.
Suboxone is supplied as a sublingual film that can either be dissolved under the tongue or inside the cheek. The guides below should be followed on how to take Suboxone either sublingually or buccally.
Sublingual Administration (under the tongue)
Buccal Administration (inside the cheek)
A burning sensation while the films are dissolving is normal, however do not take Suboxone if you are allergic to buprenorphine or naloxone. Each Suboxone prescription obtained at a pharmacy comes with a medication guide containing important information on how to use the drug.
Because Suboxone contains buprenorphine, an opioid, the user may experience side effects consistent with that of other opioids such as:
The most dangerous side effect of Suboxone is respiratory depression, defined as shallow, labored breathing. Respiratory depression is distinct from breathing problems caused by asthma or COPD. Using a rescue inhaler will not treat respiratory depression caused by an opioid overdose. Because respiratory depression can result in death if not managed quickly, it is considered a medical emergency.
In cases of respiratory depression due to Suboxone overdose, the drug Narcan is used. Narcan is a nasal spray containing naloxone, the same opioid antagonist found in Suboxone. Narcan works within minutes to restore normal breathing and is available without a prescription in most states including California.7
It is important to note that while Suboxone contains the same ingredient as Narcan, the naloxone contained in Suboxone films is not effective at treating respiratory depression, only the Narcan nasal spray should be used.
Despite the buprenorphine in Suboxone being only a partial agonist, it is an opioid nonetheless and can still make the user feel high. Consequently, Suboxone carries the potential for abuse, though not as significant as opioids such as hydrocodone, oxycodone and fentanyl.
Because individuals being treated with Suboxone are already undergoing treatment for opioid dependence, one of the most telling signs of an individual abusing Suboxone is running out of it too soon.
By law, Suboxone can only be dispensed at a pharmacy for a 30-day supply each time it is picked up. If a user takes more than what is prescribed, they will inevitably run out of Suboxone before their next refill is due. Frequent attempts to refill Suboxone early may indicate that Suboxone is being abused.
If a dose of Suboxone is missed, or the dose is reduced too quickly, a Suboxone user may experience symptoms of opioid withdrawal such as:
If Suboxone is discontinued completely, withdrawal symptoms may begin 12 hours following the last dose and peak within three days. Physical symptoms will begin to subside after 1-2 weeks, though lingering depression and drug craving may continue for weeks or months.
Suboxone should not be discontinued suddenly (“cold turkey”), as it increases the probability of experiencing withdrawal symptoms. Rather, Suboxone should be tapered by slowly decreasing the dose over time, allowing the body to adjust to lower doses of the opioid.
The timeline for treatment with Suboxone follows three main stages: induction, maintenance and tapering.
Induction – The initial period of Suboxone therapy is called induction, the goal of which is to safely suppress the symptoms of opioid withdrawal as quickly as possible with therapeutic doses of Suboxone.
Maintenance – Once induction is complete and withdrawal symptoms are adequately controlled, a maintenance period begins where a steady dose of Suboxone is taken to prevent the emergence of withdrawal symptoms.
Tapering – Once withdrawal symptoms are adequately controlled with a maintenance dose, the dose of Suboxone is tapered (reduced) over time until Suboxone can be safely discontinued.
The length of a Suboxone taper will depend on the individual. Some factors that may affect the length of a taper include:
Drug addiction is a difficult disease to manage alone; it is highly recommended that an individual addicted to opioids seek help in the management of their condition. An addiction specialist can recommend several options such as Suboxone to manage withdrawal symptoms and help along the path to stopping drug abuse.
Synthetics, also known as “designer drugs,” are completely man-made drugs created in a laboratory. Many designer drugs are created legally and serve a legitimate medical purpose, in fact most prescription drugs are either semi-synthetic or fully synthetic. In the context of illicit drugs, the term “synthetic” or “designer drug” usually refers to an illegal drug made in a clandestine laboratory.
All drugs can be classified into one of three categories – natural, semi-synthetic or synthetic.
Designer drugs are often very similar to legal or illegal controlled substances. This is because many times the production of designer drugs involves slightly tweaking the molecular structure of the already-existing “parent drug” while still retaining its effects. Since designer drugs are technically different from their parent drug, distributors attempt to sell them under the guise of research chemicals, plant food, plant material, glass cleaner, even incense or potpourri.
The Drug Enforcement Agency has identified the main source of designer drugs to be East Asia, where they are distributed at wholesale throughout Europe and the United States.
Designer drugs can be purchased online, where they are often shipped discretely directly to the buyer’s house. Buying designer drugs online is dangerous, as the authenticity of the manufacturer can rarely be confirmed.
Unlike cosmetic bath salts that are added to a relaxing bath, illicit bath salts are sold to be consumed for their psychoactive effects. They are often designed to have effects similar to stimulants such as cocaine, ecstasy and amphetamines.
Bath salts are easily purchased at gas stations, with common names including Bliss, Blue Silk, Cloud Nine, Drone, Energy-1, Ivory Wave, Burst, and Pure Ivory. These products are usually sold in 200-500 mg packets. The National Institute on Drug Abuse reports that many of these products will be labeled not for human consumption, though their manufacturers do indeed intend for them to be consumed.
The active ingredient in bath salts is nearly impossible to determine by the packaging, however they usually contain one or more synthetic cathinones, drugs with a chemical structure similar to amphetamine. The most common ingredients found in bath salts include MDPV, mephedrone and methylone. These drugs have stimulant and hallucinogenic properties while being highly addictive.
Bath salts can wreak havoc on the user’s physical and mental health. Some side effects are listed below.
Severe psychotic symptoms are common those who present to the emergency department, occurring in up to 40% of cases. Those under the influence of bath salts have an altered perception and can be extremely violent, often harming themselves or others. In one case, a man under the influence of bath salts shot himself after murdering his wife; a toxicology report identified MDPV in his system.
Synthetic cannabinoids, commonly referred to as “spice,” are man-made drugs designed to resemble THC, the primary psychoactive component in cannabis. Because spice binds to the same receptors as THC, it is sometimes referred to as “synthetic marijuana.” Some common names for synthetic cannabinoids are listed below:
Like most designer drugs, spice is manufactured overseas then shipped to the United States and Europe. It is usually dissolved in a solvent, then sprayed onto herbal products where they absorb the psychoactive ingredients. The final product, sold in gas stations or bought online, usually resembles plant material but is sometimes sold as a powder. Standard urine drug tests do not screen for spice.
Since it is nearly impossible to determine the actual ingredient in spice, the effects of any given product are difficult to predict. General effects reported include:
Heart attacks caused by spice are life-threatening. In one in case, a 19-year old who took spice collapsed from a heart attack and later died
While bath salts and spice remain the most popular designer drugs, there are others as well. Many of the following are sold online through unscrupulous retailers.
Most designer drugs come in packaging that may appear official, even feigning compliance with regulatory authorities by labeling it with statements such “not for human consumption.” Make no mistake though, most designer drugs are created in clandestine laboratories with little to no quality control in the manufacturing process. The actual amount of drug may vary from batch to batch, making it impossible to know how strong the product is.
Valium is the brand name of the prescription drug diazepam, a long-acting central nervous system depressant belonging to the benzodiazepine class of drugs. Its relaxing effect makes Valium useful to treat anxiety disorders, however many find this feeling addicting.
Tolerance to Valium develops quickly and long term use can result in both physical and psychological dependence. If stopped suddenly, a user can experience potentially life-threatening seizures.
Valium is frequently obtained with a prescription; in 2016 there were more than 6 million prescriptions in the United States.
Valium goes under the street names Vallies, Jellies, Moggies and Eggs.
Released in 1963, Valium was one of the first benzodiazepines brought to market in the United States. Benzodiazepines like Valium were originally designed to be safer alternatives to barbiturates, a class of dangerous central nervous system depressants. Despite their increased safety compared to barbiturates, benzodiazepines can still be dangerous, especially when combined with other drugs such as alcohol or opioids.
Valium exerts is calming effect by enhancing the activity of a natural “slow down” chemical in the brain called GABA (gamma-Aminobutyric acid). GABA plays an important role in how fast neurons fire. Overactive neuronal firing can result in both psychological disorders such as crippling anxiety and physical disorders such as epilepsy. By enhancing the “slow down” chemical in our brain, Valium helps those with anxiety feel more relaxed.
Valium has several legitimate medical uses including:
Valium use is associated with significant side effects. A Valium abuser may experience the following:
How strong the effects of Valium will be is dependent on several factors, including:
While Valium is usually safe when taken as prescribed, too much Valium can result in an overdose. This is especially true when taken with alcohol, which multiplies the effects of Xanax.
The signs and symptoms of Valium overdose may include the following:
While a Valium overdose is unlikely to cause death by itself, when combined with alcohol or opioids such as hydrocodone, oxycodone or morphine, overdose can be lethal. The antidote for benzodiazepine overdose is a drug called flumazenil, however it is reserved for cases of severe respiratory depression.
Many users find the relaxing effects of Valium to be addicting. While many will intentionally seek out the drug, even those without a history of substance use disorders can become addicted.
Some signs and symptoms that may signal addiction to Valium are listed below.
Tolerance to Valium develops rapidly, where more of the drug is required to achieve the same effect. When tolerance develops, users will often take more of the drug to feel high. This vicious cycle continues indefinitely, with users taking increasingly higher doses only to further build tolerance.
If taken for more than 2-3 weeks, a user may become physically dependent on Valium. Physical dependence occurs when the body adapts to the drug and eventually requires it to function properly. Once dependent, a user will suffer withdrawal if Valium is not taken.
Symptoms of withdrawal range from incredibly unpleasant to life-threatening. Some symptoms are listed below:
Seizures associated with Valium withdrawal can be life-threatening and should be managed in a hospital setting.
Because Valium stays in the body longer than most benzodiazepines, withdrawal symptoms may not begin until days following the last dose.
Addiction treatment for Valium abuse will often take place in a treatment facility, where the user can receive around-the-clock support. Medical detox in a treatment center will focus on managing both the user’s physical and mental health.
The initial goal for addiction treatment of Valium is to control withdrawal symptoms. In most cases of benzodiazepine withdrawal, the user is switched from whichever benzodiazepine they are abusing to Valium. In cases of Valium addiction, since the user is already taking Valium, the dose may be decreased (tapered) immediately to begin a tapering period.
Discontinuing Valium completely and suddenly, know as going “cold turkey,” is incredibly dangerous and should never be attempted due to the risk of life-threatening seizures. To treat Valium addiction, the dose will be slowly tapered over time.
How long the tapering period will last is very dependent on the individual, however an example taper may include decreasing the dose by 5-25% every 1-4 weeks until it can be safely discontinued. The tapering period may last months, especially for long term users who require time to recover from dose reductions.
Substance abuse is a challenging problem to overcome. Because Valium is dangerous to quit without the help of a medical professional, it is recommended that anyone addicted to Valium seek out the help of a qualified addiction specialist.